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dc.contributor.authorYoswaris Semamingen_US
dc.contributor.authorJirapas Sripetchwandeeen_US
dc.contributor.authorPiangkwan Sa-Nguanmooen_US
dc.contributor.authorHiranya Pintanaen_US
dc.contributor.authorPatchareewan Pannangpetchen_US
dc.contributor.authorNipon Chattipakornen_US
dc.contributor.authorSiriporn C. Chattipakornen_US
dc.description.abstract© 2015, National Research Council of Canada. All rights reserved Brain mitochondrial dysfunction has been demonstrated in diabetic animals with neurodegeneration. Protocatechuic acid (PCA), a major metabolite of anthocyanin, has been shown to exert glycemic control and oxidative stress reduction in the heart. However, its effects on oxidative stress and mitochondrial function in the brain under diabetic condition have never been investigated. We found that PCA exerted glycemic control, attenuates brain mitochondrial dysfunction, and contributes to the prevention of brain oxidative stress in diabetic rats.en_US
dc.subjectBiochemistry, Genetics and Molecular Biologyen_US
dc.titleProtocatechuic acid protects brain mitochondrial function in streptozotocin-induced diabetic ratsen_US
article.title.sourcetitleApplied Physiology, Nutrition and Metabolismen_US
article.volume40en_US Mai Universityen_US Universityen_US Kaen Universityen_US
Appears in Collections:CMUL: Journal Articles

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